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| Author | Topic: muerte |
| Alba |
posted 8/29/03 6:26 PM
Me siento frustrada, si todo el mundo que conozco ve la serie j&j, por qué la quitan!? Friends ya los tenemos demasiado vistos, q compaginen las dos series y no nos repitan tanto los capítulos de friends. o que me den una explicación de por qé lo kitan. lo bueno esq por lo menos hay una maldita página q me ha contado el final de la serie, nosé si para bien o para mal... bueno adiós |
| sergio |
posted 8/30/03 2:02 AM
yo pensaba que era de los poco que la veía y por eso la quitaban, pero cuando he entrado aquí en una pagina extrangera y he visto que los españoles dejaban su descontento he alucinado con que la quiten, deberiamos de hacer algo, enviar cada uno un email para que se den cuenta que hay gente que vemos la serie. Yo no he leido el final, ¿Crees que debería leerlo? |
| Alba |
posted 9/1/03 2:48 PM
hola! yo sí q me leí el final, supongo q es un poco lo que se espera, por lo que no pasa nada si lo lees, pero yo me arrepiento un poco de haberlo leído, porque.. q pasa si lo vuelven a emitir? pero bueno, gracias a q tengo poca memoria puede q no me acuerde de todo así q está bien ;) |
| Sergio |
posted 9/1/03 11:33 PM
Hola, gracias por responder, me lo he estado pensando y al final lo que he decidido es no leerlo,una razon es la que tu dices, y si vuelven a emitirla? además lo que me gusta es meterme en la historia, y es verdad lo que dices, d q pasa lo que se espera, pero creo que eso es lo bueno, particularmente me gustan los finales felices, que se corresponde con lo que esperas, no crees? así que m quedo con la esperanza de que la vuelvan a poner. Cuidate y si te apetece que hablemos más estare encantado. 1 saludo Sergio |
| nazaret |
posted 9/2/03 4:34 PM
haceis bien en no leerla. yo no me pude resistir, y mira que odio saber lo que va a pasar, pero no lo pude evitar. tu aguanta. yo temia que quitasen la serie, pero ahora se que algun dia la volveran a poner. yo he intentado escribir al plus, por si me podian dar alguna informacion, pero no me han contestado. quizas si nos unimos todos la ponen pronto. muchos saludos a todos los fans españoles de esta serie ;) |
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| Victor |
posted 1/1/05 3:29 AM
Please contribute generously to cancer research on the cancer awareness week. Thrombocytopenia may be congenital or acquired. The acquired form is more common, especially among elderly. In either case, it usually results from decreased or defective production of platelets in the bone marrow (for example, in leukemia, aplastic anemia and with toxicity from certain drugs), increased destruction outside the marrow caused by an underlying disorder (such as cirrhosis of the liver, disseminated intravascular coagulation and severe infection) or dilution of platelets. Less commonly, thrombocytopenia results from increased sequestration of platelets in the spleen. Typically, a patient with thrombocytopenia reports a sudden onset of petechiae and ecchymoses, from bleeding into mucous membranes or on the skin. Painless, round and pinpoints (1 to 3 mm in diameter), petechiae usually appear and fade, and sometimes group to form ecchymoses. Another type of blood leakage (and larger than petechiae), ecchymoses are purple, blue or yellow-green bruises that vary in size and shape. They can occur anywhere on the body from a traumatic injury. A person with thrombocytopenia may also complain of malaise, fatigue and general weakness (with or without accompanying blood loss). In acquired thrombocytopenia, the patient's history may include the use of one or several offending drugs. Inspection typically reveals evidence of bleeding (petechiae or ecchymoses), along with slow, continuous bleeding from any injuries or wounds. Adults may have large, blood-filled bullae in the mouth. If the person's platelet count is between 30,000 and 50,000/mm3, bruising with minor trauma may be expected; if it is between 15,000 and 30,000/mm3, spontaneous bruising will be seen (mostly on the arms and legs). Pathophysiology: Mature neutrophils are produced by precursors in the bone marrow. The total body neutrophil content can be divided conceptually into the following 3 compartments: the bone marrow, the blood, and the tissues. In the marrow, the neutrophils exist in 2 divisions—the proliferative, or mitotic, compartment (myeloblasts, promyelocytes, myelocytes) and the maturation-storage compartment (metamyelocytes, bands, polys). Neutrophils leave the marrow storage compartment and enter the blood without reentry into the marrow. In the blood, 2 compartments also are present, the marginal compartment and the circulating compartment. Some neutrophils do not circulate freely (marginal compartment) but are adherent to the vascular surface, and these constitute approximately half the total neutrophils in the blood compartment. Neutrophils leave the blood pool in a random manner after 6-8 hours and enter the tissues, where they are destined for cellular action or death. Thus, if the process producing neutropenia is unknown, measurements of the blood neutrophil number, ANC, often must be supplemented by bone marrow examination to determine whether adequate production of neutrophils or increased destruction of neutrophils exists. Sites and mechanisms of injury that cause neutropenia can be restricted to the mitotic or mature-storage pools in the marrow or the mature circulating pools (sequestration). Benign congenital neutropenias are associated with a decrease in circulating neutrophils but entirely normal marrow pools, marginal blood pools, and tissue neutrophils. The clinical sequelae of neutropenia manifest as infections, most commonly of the mucous membranes. Skin is the second most common infection site, manifesting as ulcers, abscesses, rashes, and delays in wound healing. The genitalia and perirectum also are affected. Signs of infection, including warmth and swelling, may be absent Treatment of thrombocytopenia varies according to the cause. If thrombocytopenia is drug-induced, then removal of the offending agents should correct the condition. Corticosteroids may be used to increase platelet production. Lithium carbonate or folate may also be used to stimulate the bone marrow production of platelets. Platelet transfusions may be used to stop episodic abnormal bleeding caused by a low platelet count. However, if platelet destruction results from an immune disorder, platelet infusions may have only a minimal effect and may be reserved for life-threatening bleeding. Splenectomy may be necessary to correct thrombocytopenia caused by platelet destruction. A splenectomy should significantly reduce platelet destruction because the spleen acts as the primary site of platelet removal and antibody production. Patients with idiopathic thrombocytopenic purpura (ITP) may require high-dose intravenous immunoglobulin. Patients with thrombotic thrombocytopenic purpura (TTP) will probably require large-volume plasmapheresis (plasma exchange). BSA calculator Thrombocytopenia Neutropenia Doxorubicin BRCA1 and BRCA2 |
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